introspection to schizophrenia Posted: 05-23-08 18:15pm
Dear Mr. Lewis A. Opler, MD, PhD,
I have been looking for someone to
have a discourse about my findings in the
field of abnormal psychology. I have been
diagnosed schizophrenic in 1997. I am an
atypical schizophrenic. I say this
because through my efforts to cognize my
own disaffect-amt to society. 1
(interpersonal) I have found that the
voices are actually "low amplitude", "high
volume", sub-threshold inter-sensory
"displays". (typically) In order, of my
cogniscence, "[by virtue of reflexive
positive reditribution et."low
amplitude"]> "high amplitude"[<as
per schizophrenic] , "high volume", at and
above threshold thyamatic experience. The
causality is a non-linear cognization with
a neupeptidic concomitance. (1*in
otherwords, I rationalized my own state of
mind)I apologize that didn't make sense.
My rationalization dosesn't conclude
properly aformentioned delivery. I should
put 1* in 1
My efforts were at first given to the
idea of an
anxyiolitic(seretonergic)-stimulant
molecule. I realized that this was not
'mobile' do to the withdrawl and
unpredictable natures of stimulants. 2
Then I was drawn to a anxyiolitic
(same)-ginsenoside . Esentially, if you
are not familiar, and anxiolytic steroid.
(to clarify [point 2] should have stated
anxyiolitic and a ginsenoside in the same
compound) I became at this point,
apropriately perplexed. I wanted the
chemical to be active at all
indeterminable levels, as per the
individual natures of those affected. So
I re-examined all the potentiating
chemicals in my repertoire, seretonin
[5HT-1a], dopaminergic, and noradrenergic.
The fact was that I didn't want a
substance that had an inhibiting nature.
So, I re-examined a thought paridigm I had
come up with to represent the
schizophrenic thought process. (given
socio-pysch-al "[tend]-isms", more
familiar to yourself as psycho-social
interaction as per trends in society) It
is:
I predicate this by saying that
neutral-behavior autoreflexive ideation is
not typically possibe. (allowing the mind
to heal itself in a voluntary way without
drugs) So we start with the gnostic
position.
1. intial knowledge defines position in
flux
2. flux defines position to decision
I submit that this is the key area where
schizophrenics would show a negative
personal affect. In the sense that people
with generally positive affect would make
choices. The former would be lent to
decisions.
1decision 1a : ... specf : the act of
settling or terminating (as a contest or
controversy) by giving judgment [1]
1choice 1: ... typically : the voluntary
and purposive or deliberate action of
picking, singalling out, or selecting from
two or more that which is favored or
superior : the decision reached by such
action <the ~ made by the voters>
<Lincoln's ~ of Grant as general>
[1]
The key point of interest,
neuropharmacologically, would be the
over-[nor?]adrenergistics with an axis
effect to the dopaminerigic pathways.
The key epidemiology involved here is how
people are introduced to schizophrenia. I
took LSD and subsequently was put in a
world of "internal chaos". I don't know
how the former experiences it. I didn't
fundamentally consider myself
schizophrenic until I had a "moment". I
thought the person on the TV was speaking
directly to me. I experienced a
'fundamental fear'. I think the former
enters into schizophrenia in this way.
The subsequent conclusion is to be
gained in the question; where is the
disassociation between neurochemical
pathways in schizophrenia and the bodies
natural anxiolytics?
3. decision evokes a perceptual change
4. perceptual change forms a stance
I submit that the stance is the
dopaminergic action. Therfore if
dopaminergic action is stance and
schizophrenia is decision-ary, then
decisions would lead to trepidation or
over-[nor?]adrenergistics. This would add
to perceptual change. Therein lies the
disassociation 'loop' [disunity] in
question.
5. stance is knowledge
I submit that if gultaminergic projections
affecting memory ...... * I was just about
to "get in deep" with the lateral
tegmental, trying to link the
noradrenergic lines with the glutamanergic
projections, as per [with] the cortex, and
I realized macroscopically, with the
exception of the greatest of collegiate
brain scientists, it is nearly impossible
to cognize "on one plate" if you will, the
mind versus the subject of interest. I
going back to a Scientific American
article.
I am still reading.... however I have
come to a former realization.... rather
that I was thinking from the philisophical
standpoint that why would God, as a
progressive compassionate intelligence
[fundamentally from most aspects providing
initial comfort {parents}, saftey
{gernerally a community or instincts}, and
the internal faculties to derive pleasure]
........ well alright... to appease all
the opinions on the subject of
existence..... free will! .....then one
has to entertain at least briefly that
schizophrenia is not a illness. I think
it would be more appropriate to have a
little introspection on the subject. I
say this because most contemporary [lay,
and I say because, it defines propensity
of culture] articles tend to disseminate
in a utilitarian fashion. The writings
hardly define the human condition, as if
the scientists had a betteer conceptual
quality of living than schizophrenics and
if it wern't for one than the other would
not survive at all, which is absurd in a
universe of all possibilites. If we can't
be dreamers alone then how can existence
satiable?
Going on with my diatribe, if you will
.... it's only been
about 5 minutes or so. I think it's a
fundamental culture thing. That's why I
am a proponent of the steroid aspect of
treatment.
I need to read on a get some more info ,
state of mind..
Sincerely,
Darius Leshko Slaski
[1] (Webster's Third New International
Dictionary)
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DariusS
New User, Becoming EHEALTHy
Joined: 23 May 2008 Posts: 2
introspection to schizophrenia Posted: 05-23-08 18:38pm
I recently also have been interested in
the aspect that the visual processing
center is anterior to the Brocas area. As
in LSD, one tends to see things. Also
interestingly as in neonates, sertetonin
as a greater compared with adults where
dopamine tends to be prominent, this is
the time of imprintment. I would say that
seretonin has a heavy role in memory given
the right neurochemical circumstances.
Keeping this in mind, consider also that
of people with synesthesia. I submit a
possibility that schizophrenia or atleast
drug induced toxic psychosis leading to
schizophrenia, is chemical induced "lift"
and seretonergic "placement". The fact by
which the mind is introduced to a vivid
field of visions in concomitance to a high
level of seretonergic activity and by
which through the extended period of
interaction 8-12hours is a state of
prefered visual interaction, contrary to
the dopamerigic interactions, pleasurable,
commonly attributable with speech.
(consider the propensity to introvertism
during the experience and preoccupation
with awarness)