Cerebro-spinal liquid (CSL) is produced by
the plexus choroideus found in the I
(first) and II brain chamber. From here,
CSL goes into the III and IV brain
chamber. The IV chamber communicates with
the subarachnoidal space via 2 anatomic
openings. The subarachnoidal space is a
space between the arachnoidal and pia
covering of the brain and spinal cord.
From the subarachnoidal space through the
arachnoidal, CSL enters the venous sinuses
created by the dural covering of the
brain. Venous sinuses collect the entire
venous blood stream with CSL from the
brain and transport them to the heart.
A" hydrocephalus" indicates increased
volume and pressure of CSL followed by
enlargement of the brain chambers. An
enlarged brain chamber then causes atrophy
of brain tissue. Any stop in liquid
circulation and drainage from the chambers
and subarachnoidal space can cause a
corresponding increase in CSL-pressure and
volume. A blockage of CSL circulation can
be due to congenital anomalies, tumors,
consequences from infections and traumas
etc. Increased pressure of the
cerebro-spinal liquid (CSL) can be also
due to increased production of CSL in the
brain chambers and subarachnoidal space.
There is increased volume of CSL in cases
of inflammation (meningitis or
encephalitis), subarachnoidal hemorrhage
(bleeding in subarachnoidal space),
intra-ventricular bleeding (bleeding in
brain’s chambers), etc.
Short term memory loss can be due to
psychiatric or neurological disorders.
Anxiety and dementia are also frequent
reasons for short term memory loss.
Dementia can be due to many reasons
including hydrocephalus. In most cases of
increased CSL pressure, however, “short
term memory loss” is not a dominant
symptom.
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